I said it was a possible treatment, not a cure.
As another Lame Cherry exclusive in matter anti matter.
The Lame Cherry is passing along an interesting paper on the use of some common antibiotics and anti malarial and parasite drugs in use to inhibit prions.
I found it interesting in when I was so sick with another satanic attack on my person, what I took was a cheap antibiotic in doxycycline. I told the doctor this, he puzzled for a moment and said that it should not be working, but as it was, he was going to prescribe it.
My condition was inflammation to allergens in cheese of all things. There does seem to be a thread in this of these yeasts, fungus, parasite and prions all having a connective tissue,and all of the above have been rampant in humans the past generation and growing, in being diagnosed as something else.
This is just part of the list as I do not believe this is a cure. It does seem to inhibit to increase survival time, but the reason that spike protein was chosen is because it is so universal as a germ delivery that it can not be improved upon.
Doxycycline and Minocycline Two closely related tetracycline antibiotics, doxycycline and minocycline, have shown signs of efficacy against prion disease. Doxycycline can penetrate the blood brain barrier [24]. This antibiotic blocks tau amyloid aggregation and its associated neuronal toxicity in vitro [25,26]. Doxycycline can inhibit the aggregation of Aβ42 amyloid fibrils and disassemble mature amyloid fibrils [27]. Doxycycline blocks alpha synuclein aggregation in cell culture [28] and benefits were seen in a mouse model [29]. Doxycycline is effective in a mouse model of Huntington’s disease [30] as well as a mouse model of insomnia [31]. The clinical trial results with doxycycline have been mixed. In a clinical trial reported by Varges [32] doxycycline was administered to early stage patients with CJD and showed a benefit in survival. Not unexpectedly when doxycycline was administered in a clinical trial to patients with late stage disease, there was no benefit [33]. Minocycline is a tetracycline antibiotic like doxycycline with many similar properties. There are however, some reports that minocycline has special neuroprotective activity not seen with doxycycline [34]. Minocycline has been shown to have multiple effects that inhibit neurodegeneration [35]. These include effects on protein misfolding, inhibiting neuroinflammation, scavenging free radicals, mitochondrial cytoprotection, as well as altering both proteolysis and apoptosis [36]. The effect of minocycline on prion diseases [37] and Parkinson’s disease has been reviewed [38].
Quinacrine, an antimalarial drug similar to hydroxychloroquine, has been shown to have some efficacy in prion disease as discussed below. There is also data it is efficacious in treating COVID-19 [39] in part due to binding to the RNA of the coronavirus causing COVID-19. This mechanism would also be expected to prevent the development of some prion diseases caused directly by the mRNA sequence in the COVID “vaccines” [1]. Mossad linked groups, the World Health Organization (WHO) for example, have promoted a false narrative attempting to dissuade people with COVID-19 from using related antimalarial drugs, hydroxychloroquine and chloroquine, for treating COVID-19. This narrative raises suspicions that quinacrine may also be useful in COVID-19 “vaccine” induced prion disease. Quinacrine crosses the blood brain barrier at least in mice [40]. Quinacrine has been shown in vitro to have properties that may be beneficial in prion disease. Quinacrine binds to some prion proteins. According to one paper [14] “These results confirm that quinacrine almost exclusively reacts with the thiol groups present in proteins and peptides. The chemical reaction alters the prion properties and increases the concentration of the acridine moiety in the prion protein.” Furthermore, quinacrine directly dissociated amyloid plaques in brains of transgenic mice [42]. Quinacrine may inhibit stimuli from causing proteins from moving into their prion formation [43]. Like doxycycline, published clinical trial literature on quinacrine has mixed results. Quinacrine showed some benefit in an mostly open label study of prion patients in the UK [44]. Quinacrine failed a 2 month randomized study of 54 CJD patients [45]. However, those trial participants who received quinacrine for two months during the randomized phase and received prolonged quinacrine as an open label extension had impressively increased survival than those who did not receive the extended treatment of quinacrine. Taken together the combined studies [44,45] indicate that quinacrine may have substantial benefit if given early in disease and taken for a prolonged period of time. However, the drug is not considered as safe as doxycycline. Ivermectin There is little data on the use of ivermectin to treat prion disease per se. There is however data on its efficacy to treat COVID-19 infections [46,47]. The author’s interest in this drug relates to the loud false narrative from Mossad associated organizations to dissuade people from using ivermectin for COVID-19 infections [48]. Ivermectin has the ability to bind the prion inducing spike protein at its receptor-binding domain, preventing the spike protein from entering a cell [49] and inducing prion disease
Nuff Said
agtG
